Wednesday, May 16, 2012
Trigeminal neuralgia: Assessment of neurovascular decompression by 3D fast imaging employing steady-
Ruth Prieto, José M Pascual, Miguel Yus, Manuela Jorquera
Surgical Neurology International 2012 3(1):50-50
Background: Trigeminal neuralgia is most commonly caused by vascular compression at the trigeminal nerve (TN) root entry zone. Microvascular decompression (MVD) has been established as a useful treatment. Outcome depends on the correct identification of the compression site and its adequate decompression at surgery. Preoperative identification of neurovascular compression might predict which patients will benefit from MVD. Management of persistent or recurrent trigeminal neuralgia after an MVD is a baffling problem for neurosurgeons. An accurate neuroradiological evaluation of the TN padding following a failed MVD might help identify the underlying cause and plan further treatment. Case description: A 68-year-old female presented with a right-sided trigeminal neuralgia (V3) refractory to medical therapy. A high-resolution three-dimensional magnetic resonance imaging (3D MRI) study included fast imaging employing steady-state acquisition (FIESTA) and time of flight multiple overlapping thin slab acquisition (TOF MOTSA) sequences to evaluate the neurovascular anatomy in the cerebellopontine angle. An unambiguous compression of the right TN at the rostral-medial site by the superior cerebellar artery (SCA) was identified. The SCA loop compressing the TN was identical in location and configuration to that predicted in the preoperative study. After the MVD, the patient was relieved from her pain and a postoperative high-resolution 3D MRI study confirmed the appropriate placement of the Teflon implant between the TN and SCA. Conclusion: To our knowledge, this is the first report that characterizes the proper TN padding by high-resolution 3D MRI after trigeminal MVD. The present case also emphasizes the importance of performing a 3D MRI in patients with trigeminal neuralgia to anticipate the surgeon's view and predict the outcome after MVD.
Surgical Neurology International 2012 3(1):50-50
Background: Trigeminal neuralgia is most commonly caused by vascular compression at the trigeminal nerve (TN) root entry zone. Microvascular decompression (MVD) has been established as a useful treatment. Outcome depends on the correct identification of the compression site and its adequate decompression at surgery. Preoperative identification of neurovascular compression might predict which patients will benefit from MVD. Management of persistent or recurrent trigeminal neuralgia after an MVD is a baffling problem for neurosurgeons. An accurate neuroradiological evaluation of the TN padding following a failed MVD might help identify the underlying cause and plan further treatment. Case description: A 68-year-old female presented with a right-sided trigeminal neuralgia (V3) refractory to medical therapy. A high-resolution three-dimensional magnetic resonance imaging (3D MRI) study included fast imaging employing steady-state acquisition (FIESTA) and time of flight multiple overlapping thin slab acquisition (TOF MOTSA) sequences to evaluate the neurovascular anatomy in the cerebellopontine angle. An unambiguous compression of the right TN at the rostral-medial site by the superior cerebellar artery (SCA) was identified. The SCA loop compressing the TN was identical in location and configuration to that predicted in the preoperative study. After the MVD, the patient was relieved from her pain and a postoperative high-resolution 3D MRI study confirmed the appropriate placement of the Teflon implant between the TN and SCA. Conclusion: To our knowledge, this is the first report that characterizes the proper TN padding by high-resolution 3D MRI after trigeminal MVD. The present case also emphasizes the importance of performing a 3D MRI in patients with trigeminal neuralgia to anticipate the surgeon's view and predict the outcome after MVD.
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Clinical outcomes of myelomeningocele defect closure over 10 years
Publication year: 2012
Source:Journal of Clinical Neuroscience
Ahmet Murat Müslüman, Semra Karşıdağ, Deniz Özgür Sucu, Arzu Akçal, Adem Yılmaz, Deniz Şirinoğlu, Yunus Aydın
We report our surgical procedures for the closure of myelomeningocele defects. A retrospective analysis of 162 patients (74 male [45.7%], 88 female [54.3%]) with myelomeningocele was performed and the relationship between hydrocephalus, neurological status and the level and size of the myelomeningocele was described according to type of defect closure. Patients were divided into four groups according to the size of the defect, which was classified into ranges of 0–24cm2, 25–39cm2, 40–60cm2 and >60cm2. Myelomeningocele occurred in the lumbar region in 114 patients (70%). The minimum defect size was 3×2cm, and the maximum defect size was 15×15cm (mean defect size=34.64cm2). We found that primary closure can be performed on clean, small defects with an intact sac that contains cerebrospinal fluid and the neural placode. For defects larger than 25cm2 that contained perforated sacculas, more soft tissue for well-vascularized coverage was required. Bilateral V–Y fasciocutaneous flaps are a good choice for immediate coverage of myelomeningocele defects.
Source:Journal of Clinical Neuroscience
Ahmet Murat Müslüman, Semra Karşıdağ, Deniz Özgür Sucu, Arzu Akçal, Adem Yılmaz, Deniz Şirinoğlu, Yunus Aydın
We report our surgical procedures for the closure of myelomeningocele defects. A retrospective analysis of 162 patients (74 male [45.7%], 88 female [54.3%]) with myelomeningocele was performed and the relationship between hydrocephalus, neurological status and the level and size of the myelomeningocele was described according to type of defect closure. Patients were divided into four groups according to the size of the defect, which was classified into ranges of 0–24cm2, 25–39cm2, 40–60cm2 and >60cm2. Myelomeningocele occurred in the lumbar region in 114 patients (70%). The minimum defect size was 3×2cm, and the maximum defect size was 15×15cm (mean defect size=34.64cm2). We found that primary closure can be performed on clean, small defects with an intact sac that contains cerebrospinal fluid and the neural placode. For defects larger than 25cm2 that contained perforated sacculas, more soft tissue for well-vascularized coverage was required. Bilateral V–Y fasciocutaneous flaps are a good choice for immediate coverage of myelomeningocele defects.
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iPad gives voice to kids with autism
Sharia stood immobile in front of the television, transfixed by its images, unaware of the world around her. Her family called her name over and over again, but she did not respond. It was that moment when they knew something was wrong.
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Surgical application of smartphones
Blackouts are common in Greek public hospitals.1 When lights go off in the operating room, during a surgical procedure, the situation is understandably dramatic.A Greek obstetrician at the public...
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